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diff quik stain set  (Dade Behring)

 
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    Dade Behring diff quik stain set
    Diff Quik Stain Set, supplied by Dade Behring, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/diff+quik+staining+set/diff+quick/pmc12635137-130-10-13
    Average 86 stars, based on 1 article reviews
    diff quik stain set - by Bioz Stars, 2026-10
    86/100 stars

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    Related Articles

    Membrane:

    Article Title: Epidermal Growth Factor Receptor Mediates Increased Cell Proliferation, Migration, and Aggregation in Esophageal Keratinocytes in Vitro and in Vivo
    Article Snippet: .. Cells that migrated through the membrane and attached to the bottom of the membrane were fixed and stained with reagents from the Diff Quik staining set (Dade Behring, Newark, DE). ..

    Article Title: EGFR-induced cell migration is mediated predominantly by the JAK-STAT pathway in primary esophageal keratinocytes.
    Article Snippet: Andl, Claudia D., Takaaki Mizushima, Kenji Oyama, Mark Bowser, Hiroshi Nakagawa, and Anil K. Rustgi.. EGFR-induced cell migration is mediated predominantly by the JAK-STAT pathway in primary esophageal keratinocytes.. Am J Physiol Gastrointest Liver Physiol 287: G1227–G1237, 2004.

    Staining:

    Article Title: Epidermal Growth Factor Receptor Mediates Increased Cell Proliferation, Migration, and Aggregation in Esophageal Keratinocytes in Vitro and in Vivo
    Article Snippet: .. Cells that migrated through the membrane and attached to the bottom of the membrane were fixed and stained with reagents from the Diff Quik staining set (Dade Behring, Newark, DE). ..

    Article Title: Prolonged Drug Selection of Breast Cancer Cells and Enrichment of Cancer Stem Cell Characteristics
    Article Snippet: .. Cell culture inserts were then placed into a Diff Quik staining set (Dade Behring, Newark, DE) and treated with fixative (triarylmethane and methanol, 1:1 vol/vol) for 2 minutes, with stain 1 (xanthenes dye) for 2 minutes, with stain 2 (thiazine dye) for 2 minutes, and then with two 2-minute water washes. ..

    Article Title: EGFR-induced cell migration is mediated predominantly by the JAK-STAT pathway in primary esophageal keratinocytes.
    Article Snippet: Andl, Claudia D., Takaaki Mizushima, Kenji Oyama, Mark Bowser, Hiroshi Nakagawa, and Anil K. Rustgi.. EGFR-induced cell migration is mediated predominantly by the JAK-STAT pathway in primary esophageal keratinocytes.. Am J Physiol Gastrointest Liver Physiol 287: G1227–G1237, 2004.

    Diff-Quik:

    Article Title: Epidermal Growth Factor Receptor Mediates Increased Cell Proliferation, Migration, and Aggregation in Esophageal Keratinocytes in Vitro and in Vivo
    Article Snippet: .. Cells that migrated through the membrane and attached to the bottom of the membrane were fixed and stained with reagents from the Diff Quik staining set (Dade Behring, Newark, DE). ..

    Article Title: Prolonged Drug Selection of Breast Cancer Cells and Enrichment of Cancer Stem Cell Characteristics
    Article Snippet: .. Cell culture inserts were then placed into a Diff Quik staining set (Dade Behring, Newark, DE) and treated with fixative (triarylmethane and methanol, 1:1 vol/vol) for 2 minutes, with stain 1 (xanthenes dye) for 2 minutes, with stain 2 (thiazine dye) for 2 minutes, and then with two 2-minute water washes. ..

    Article Title: EGFR-induced cell migration is mediated predominantly by the JAK-STAT pathway in primary esophageal keratinocytes.
    Article Snippet: Andl, Claudia D., Takaaki Mizushima, Kenji Oyama, Mark Bowser, Hiroshi Nakagawa, and Anil K. Rustgi.. EGFR-induced cell migration is mediated predominantly by the JAK-STAT pathway in primary esophageal keratinocytes.. Am J Physiol Gastrointest Liver Physiol 287: G1227–G1237, 2004.

    Cell Culture:

    Article Title: Prolonged Drug Selection of Breast Cancer Cells and Enrichment of Cancer Stem Cell Characteristics
    Article Snippet: .. Cell culture inserts were then placed into a Diff Quik staining set (Dade Behring, Newark, DE) and treated with fixative (triarylmethane and methanol, 1:1 vol/vol) for 2 minutes, with stain 1 (xanthenes dye) for 2 minutes, with stain 2 (thiazine dye) for 2 minutes, and then with two 2-minute water washes. ..



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    Anti-asthmatic effect of oleic acid according to BALF and serum IgE analysis and histopathological observation. The populations of (A) WBCs and (B) eosinophils (Eos) in the bronchoalveolar lavage fluid (BALF) were elevated due to OVA exposure and subsequently reduced by oleic acid treatment. Notably, oleic acid reduced the number of white blood cells and eosinophils in a concentration-dependent manner (n = 4). (C) Observation of inflammatory cells in BALF through Diff <t>Quik</t> <t>staining</t> showed a dose-dependent decrease with oleic acid treatment. Scale bar, 100 μm; magnification, 400×. (D) Serum IgE levels, which were elevated in the OVA-induced group, decreased in a dose-dependent manner with oleic acid treatment at various concentrations (n = 8). (E) Lung morphology, characterized by hypersecretion of mucus, proliferation of epithelial cells, and infiltration of inflammatory cells around the bronchoalveolar ducts and blood vessels, which were altered due to OVA, improved with oleic acid treatment in a dose-dependent manner. Scale bar, 100 μm; magnification, × 200. (F) Bronchoalveolar mucus secretion induced by OVA was reduced by treatment with varying concentrations of oleic acid (in a dose-dependent manner). Scale bar, 100 μm; magnification, × 200. a, CON; b, OVA; c, 1 mg/kg DEX; d, 50 mg/kg oleic acid; e, 125 mg/kg oleic acid; f, 250 mg/kg oleic acid. The results are expressed as the mean ± standard deviation. * p < 0.05 vs. CON; ** p < 0.001 vs. CON; $ p < 0.05 vs. OVA; $$ p < 0.001 vs. OVA; # p < 0.05 vs. 1 mg/kg DEX; ## p < 0.001 vs. 1 mg/kg DEX; @ p < 0.05 vs. 50 mg/kg oleic acid; @@ p < 0.001 vs. 50 mg/kg oleic acid; & p < 0.05 vs. 125 mg/kg oleic acid.
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    Anti-asthmatic effect of oleic acid according to BALF and serum IgE analysis and histopathological observation. The populations of (A) WBCs and (B) eosinophils (Eos) in the bronchoalveolar lavage fluid (BALF) were elevated due to OVA exposure and subsequently reduced by oleic acid treatment. Notably, oleic acid reduced the number of white blood cells and eosinophils in a concentration-dependent manner (n = 4). (C) Observation of inflammatory cells in BALF through Diff <t>Quik</t> <t>staining</t> showed a dose-dependent decrease with oleic acid treatment. Scale bar, 100 μm; magnification, 400×. (D) Serum IgE levels, which were elevated in the OVA-induced group, decreased in a dose-dependent manner with oleic acid treatment at various concentrations (n = 8). (E) Lung morphology, characterized by hypersecretion of mucus, proliferation of epithelial cells, and infiltration of inflammatory cells around the bronchoalveolar ducts and blood vessels, which were altered due to OVA, improved with oleic acid treatment in a dose-dependent manner. Scale bar, 100 μm; magnification, × 200. (F) Bronchoalveolar mucus secretion induced by OVA was reduced by treatment with varying concentrations of oleic acid (in a dose-dependent manner). Scale bar, 100 μm; magnification, × 200. a, CON; b, OVA; c, 1 mg/kg DEX; d, 50 mg/kg oleic acid; e, 125 mg/kg oleic acid; f, 250 mg/kg oleic acid. The results are expressed as the mean ± standard deviation. * p < 0.05 vs. CON; ** p < 0.001 vs. CON; $ p < 0.05 vs. OVA; $$ p < 0.001 vs. OVA; # p < 0.05 vs. 1 mg/kg DEX; ## p < 0.001 vs. 1 mg/kg DEX; @ p < 0.05 vs. 50 mg/kg oleic acid; @@ p < 0.001 vs. 50 mg/kg oleic acid; & p < 0.05 vs. 125 mg/kg oleic acid.
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    Anti-asthmatic effect of oleic acid according to BALF and serum IgE analysis and histopathological observation. The populations of (A) WBCs and (B) eosinophils (Eos) in the bronchoalveolar lavage fluid (BALF) were elevated due to OVA exposure and subsequently reduced by oleic acid treatment. Notably, oleic acid reduced the number of white blood cells and eosinophils in a concentration-dependent manner (n = 4). (C) Observation of inflammatory cells in BALF through Diff <t>Quik</t> <t>staining</t> showed a dose-dependent decrease with oleic acid treatment. Scale bar, 100 μm; magnification, 400×. (D) Serum IgE levels, which were elevated in the OVA-induced group, decreased in a dose-dependent manner with oleic acid treatment at various concentrations (n = 8). (E) Lung morphology, characterized by hypersecretion of mucus, proliferation of epithelial cells, and infiltration of inflammatory cells around the bronchoalveolar ducts and blood vessels, which were altered due to OVA, improved with oleic acid treatment in a dose-dependent manner. Scale bar, 100 μm; magnification, × 200. (F) Bronchoalveolar mucus secretion induced by OVA was reduced by treatment with varying concentrations of oleic acid (in a dose-dependent manner). Scale bar, 100 μm; magnification, × 200. a, CON; b, OVA; c, 1 mg/kg DEX; d, 50 mg/kg oleic acid; e, 125 mg/kg oleic acid; f, 250 mg/kg oleic acid. The results are expressed as the mean ± standard deviation. * p < 0.05 vs. CON; ** p < 0.001 vs. CON; $ p < 0.05 vs. OVA; $$ p < 0.001 vs. OVA; # p < 0.05 vs. 1 mg/kg DEX; ## p < 0.001 vs. 1 mg/kg DEX; @ p < 0.05 vs. 50 mg/kg oleic acid; @@ p < 0.001 vs. 50 mg/kg oleic acid; & p < 0.05 vs. 125 mg/kg oleic acid.
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    Anti-asthmatic effect of oleic acid according to BALF and serum IgE analysis and histopathological observation. The populations of (A) WBCs and (B) eosinophils (Eos) in the bronchoalveolar lavage fluid (BALF) were elevated due to OVA exposure and subsequently reduced by oleic acid treatment. Notably, oleic acid reduced the number of white blood cells and eosinophils in a concentration-dependent manner (n = 4). (C) Observation of inflammatory cells in BALF through Diff <t>Quik</t> <t>staining</t> showed a dose-dependent decrease with oleic acid treatment. Scale bar, 100 μm; magnification, 400×. (D) Serum IgE levels, which were elevated in the OVA-induced group, decreased in a dose-dependent manner with oleic acid treatment at various concentrations (n = 8). (E) Lung morphology, characterized by hypersecretion of mucus, proliferation of epithelial cells, and infiltration of inflammatory cells around the bronchoalveolar ducts and blood vessels, which were altered due to OVA, improved with oleic acid treatment in a dose-dependent manner. Scale bar, 100 μm; magnification, × 200. (F) Bronchoalveolar mucus secretion induced by OVA was reduced by treatment with varying concentrations of oleic acid (in a dose-dependent manner). Scale bar, 100 μm; magnification, × 200. a, CON; b, OVA; c, 1 mg/kg DEX; d, 50 mg/kg oleic acid; e, 125 mg/kg oleic acid; f, 250 mg/kg oleic acid. The results are expressed as the mean ± standard deviation. * p < 0.05 vs. CON; ** p < 0.001 vs. CON; $ p < 0.05 vs. OVA; $$ p < 0.001 vs. OVA; # p < 0.05 vs. 1 mg/kg DEX; ## p < 0.001 vs. 1 mg/kg DEX; @ p < 0.05 vs. 50 mg/kg oleic acid; @@ p < 0.001 vs. 50 mg/kg oleic acid; & p < 0.05 vs. 125 mg/kg oleic acid.
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    Image Search Results


    Anti-asthmatic effect of oleic acid according to BALF and serum IgE analysis and histopathological observation. The populations of (A) WBCs and (B) eosinophils (Eos) in the bronchoalveolar lavage fluid (BALF) were elevated due to OVA exposure and subsequently reduced by oleic acid treatment. Notably, oleic acid reduced the number of white blood cells and eosinophils in a concentration-dependent manner (n = 4). (C) Observation of inflammatory cells in BALF through Diff Quik staining showed a dose-dependent decrease with oleic acid treatment. Scale bar, 100 μm; magnification, 400×. (D) Serum IgE levels, which were elevated in the OVA-induced group, decreased in a dose-dependent manner with oleic acid treatment at various concentrations (n = 8). (E) Lung morphology, characterized by hypersecretion of mucus, proliferation of epithelial cells, and infiltration of inflammatory cells around the bronchoalveolar ducts and blood vessels, which were altered due to OVA, improved with oleic acid treatment in a dose-dependent manner. Scale bar, 100 μm; magnification, × 200. (F) Bronchoalveolar mucus secretion induced by OVA was reduced by treatment with varying concentrations of oleic acid (in a dose-dependent manner). Scale bar, 100 μm; magnification, × 200. a, CON; b, OVA; c, 1 mg/kg DEX; d, 50 mg/kg oleic acid; e, 125 mg/kg oleic acid; f, 250 mg/kg oleic acid. The results are expressed as the mean ± standard deviation. * p < 0.05 vs. CON; ** p < 0.001 vs. CON; $ p < 0.05 vs. OVA; $$ p < 0.001 vs. OVA; # p < 0.05 vs. 1 mg/kg DEX; ## p < 0.001 vs. 1 mg/kg DEX; @ p < 0.05 vs. 50 mg/kg oleic acid; @@ p < 0.001 vs. 50 mg/kg oleic acid; & p < 0.05 vs. 125 mg/kg oleic acid.

    Journal: Frontiers in Immunology

    Article Title: Oleic acid attenuates asthma pathogenesis via Th1/Th2 immune cell modulation, TLR3/4-NF-κB-related inflammation suppression, and intrinsic apoptotic pathway induction

    doi: 10.3389/fimmu.2024.1429591

    Figure Lengend Snippet: Anti-asthmatic effect of oleic acid according to BALF and serum IgE analysis and histopathological observation. The populations of (A) WBCs and (B) eosinophils (Eos) in the bronchoalveolar lavage fluid (BALF) were elevated due to OVA exposure and subsequently reduced by oleic acid treatment. Notably, oleic acid reduced the number of white blood cells and eosinophils in a concentration-dependent manner (n = 4). (C) Observation of inflammatory cells in BALF through Diff Quik staining showed a dose-dependent decrease with oleic acid treatment. Scale bar, 100 μm; magnification, 400×. (D) Serum IgE levels, which were elevated in the OVA-induced group, decreased in a dose-dependent manner with oleic acid treatment at various concentrations (n = 8). (E) Lung morphology, characterized by hypersecretion of mucus, proliferation of epithelial cells, and infiltration of inflammatory cells around the bronchoalveolar ducts and blood vessels, which were altered due to OVA, improved with oleic acid treatment in a dose-dependent manner. Scale bar, 100 μm; magnification, × 200. (F) Bronchoalveolar mucus secretion induced by OVA was reduced by treatment with varying concentrations of oleic acid (in a dose-dependent manner). Scale bar, 100 μm; magnification, × 200. a, CON; b, OVA; c, 1 mg/kg DEX; d, 50 mg/kg oleic acid; e, 125 mg/kg oleic acid; f, 250 mg/kg oleic acid. The results are expressed as the mean ± standard deviation. * p < 0.05 vs. CON; ** p < 0.001 vs. CON; $ p < 0.05 vs. OVA; $$ p < 0.001 vs. OVA; # p < 0.05 vs. 1 mg/kg DEX; ## p < 0.001 vs. 1 mg/kg DEX; @ p < 0.05 vs. 50 mg/kg oleic acid; @@ p < 0.001 vs. 50 mg/kg oleic acid; & p < 0.05 vs. 125 mg/kg oleic acid.

    Article Snippet: Differential cell counts were performed on the resuspended BALF white blood cells (WBCs) using a Hemavet Multispecies Hematology System (Drew Scientific, Waterbury, CT, USA) and the Diff Quik staining set (Thermo Fisher Scientific).

    Techniques: Concentration Assay, Diff-Quik, Staining, Standard Deviation